Fig. 7From: Dietary methionine deficiency stunts growth and increases fat deposition via suppression of fatty acids transportation and hepatic catabolism in Pekin ducksSummary of the differentially expressed proteins involved in lipid metabolism induced by dietary Met deficiency. Proteins involved in lipid metabolism that were affected by dietary Met deficiency; those in red were up-expressed, whereas those in green were down-expressed. FAs, Fatty acids; TG, Triglyceride; ALB, Albumin; FA-CoA, Acyl-CoA; A-CoA, Acetyl-CoA; KB, Ketone body; PA, Pyruvic acid; LD, Lactic acid; GLU, glucose; NEFA, Non-esterified fatty acid; LDH, Lactic dehydrogenase; L-FABP (FABP1), Fatty acid-binding protein, liver; ACBP, Acyl-CoA-binding protein; ACSL5, Acyl coenzyme A long-chain 5 synthetase; FATP5, Fatty acid transport protein 5; ATGL, Adipose triacylglyceride lipase; ACADM, Acyl-CoA dehydrogenase medium chain; NDUFS1, NADH:ubiquinone oxidoreductase core subunit S1; HSL, Hormone-sensitive lipase; ATP, Adenosine Triphosphate; MDH, Malate dehydrogenase; IDH1, Isocitrate dehydrogenase; DLD, Dihydrolipoyl dehydrogenase; HADH, Hydroxyacyl-CoA dehydrogenase; GAPDH, Glyceraldehyde-3-phosphate dehydrogenase; PGK1, Phosphoglycerate kinase; HMGCS2, 3-hydroxy-3-methylglutaryl coenzyme A synthaseBack to article page