Figure 5From: Regulation of protein degradation pathways by amino acids and insulin in skeletal muscle of neonatal pigsThe abundance and phosphorylation of ULK1 in longissimus dorsi muscle in response to age and amino acids, insulin, and leucine infusion. (A) Muscle ULK1 abundance from experiment 1 during euinsulinemic-euglycemic-euaminoacidemic (control; C), euinsulinemic-euglycemic-hyperaminoacidemic (AA), and hyperinsulinemic-euglycemic-euaminoacidemic (INS) clamps for 2 h in 6- and 26-d-old pigs. (B) Muscle ULK1 abundance from experiment 2 during euinsulinemic-euglycemic-euaminoacidemic-euleucinemic (C), euinsulinemic-euglycemic-hypoaminoacidemic-hyperleucinemic (L), and euinsulinemic-euglycemic-euaminoacidemic-hyperleucinemic (L+AA) clamps for 24 h in 5-d-old pigs. (C) Muscle ULK1 phosphorylation from experiment 1 during C, AA, and INS clamps for 2 h in 6- and 26-d-old pigs. (D) Muscle ULK1 phosphorylation from experiment 2 during C, L, and L+AA clamps for 24 h in 5-d-old pigs. Data are expressed in arbitrary units (AU). Values are means ± SEM, n = 4–7. Values not sharing common symbols differ significantly (P < 0.05).Back to article page